Search results for "modes of action"

showing 5 items of 5 documents

Editorial: Mesenchymal Stromal Cell Therapy for Regenerative Medicine

2022

Mesenchymal stromal/stem cell (MSC) therapies are increasingly explored as novel regenerative and immunomodulatory approaches to treat or prevent diseases (Pittenger et al., 2019; Hmadcha et al., 2020; Moll et al., 2020b; Ringdén et al., 2022). These cells exhibit potent paracrine properties that can modulate host immune responses, lower inflammation, and orchestrate endogenous tissue repair, at both the local and the systemic level through multiple pathways (Singer and Caplan, 2011; Doorn et al., 2012). MSCs possess tropism toward damaged and inflamed tissues, where they can engraft short-term and exert their therapeutic effects by both direct and indirect mechanisms (Doorn et al., 2012; G…

ImmunomodulationCellular and Molecular NeuroscienceModes of action (MoA)Mesenchymal stromal/stem cell (MSC)Cèl·lulesClinical translationNeurociènciesRegeneration
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Genetic Variability of Hepatitis C Virus before and after Combined Therapy of Interferon plus Ribavirin

2008

We present an analysis of the selective forces acting on two hepatitis C virus genome regions previously postulated to be involved in the viral response to combined antiviral therapy. One includes the three hypervariable regions in the envelope E2 glycoprotein, and the other encompasses the PKR binding domain and the V3 domain in the NS5A region. We used a cohort of 22 non-responder patients to combined therapy (interferon alpha-2a plus ribavirin) for which samples were obtained before initiation of therapy and after 6 or/and 12 months of treatment. A range of 25-100 clones per patient, genome region and time sample were sequenced. These were used to detect general patterns of adaptation, t…

Genome evolutionHepatitis C virusEvolutionary Biology/Bioinformaticslcsh:MedicineAlpha interferonGenome ViralHepacivirusBiologyVirology/Immune EvasionInterferon alpha-2Viral Nonstructural Proteinsmedicine.disease_causeGenomeAntiviral AgentsEvolution Molecularchemistry.chemical_compoundGenetics and Genomics/Population GeneticsRibavirinmedicineHumanslcsh:ScienceNS5APhylogenyGenetics:CIENCIAS DE LA VIDA::Genética ::Otras [UNESCO]Virology/Antivirals including Modes of Action and ResistanceMultidisciplinaryEvolutionary Biology/Evolutionary and Comparative GeneticsHepatitis C virusRibavirinlcsh:RGenetic VariationInterferon-alphaVirologyComplementarity Determining RegionsHepatitis CVirology/Virus Evolution and SymbiosisRecombinant ProteinsUNESCO::CIENCIAS DE LA VIDA::Genética ::OtrasHypervariable regionchemistryViral evolutionInterferonlcsh:QGenetic variabilityHepatitis C virus; Genetic variability; Interferon; Ribavirin; Combined therapyCombined therapyResearch ArticlePLoS ONE
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Baseline prediction of combination therapy outcome in hepatitis C virus 1b infected patients by discriminant analysis using viral and host factors.

2010

Background Current treatment of chronic hepatitis C virus (HCV) infection has limited efficacy −especially among genotype 1 infected patients−, is costly, and involves severe side effects. Thus, predicting non-response is of major interest for both patient wellbeing and health care expense. At present, treatment cannot be individualized on the basis of any baseline predictor of response. We aimed to identify pre-treatment clinical and virological parameters associated with treatment failure, as well as to assess whether therapy outcome could be predicted at baseline. Methodology Forty-three HCV subtype 1b (HCV-1b) chronically infected patients treated with pegylated-interferon alpha plus ri…

MaleHepaciviruslcsh:MedicineHepacivirusmedicine.disease_causePolyethylene Glycolschemistry.chemical_compoundlcsh:ScienceMultidisciplinarybiologyDiscriminant AnalysisHepatitis CMiddle AgedViral LoadPrognosisHepatitis CRecombinant ProteinsTreatment OutcomeGastroenterology and Hepatology/Gastrointestinal InfectionsDrug Therapy CombinationFemaleViral hepatitisViral loadResearch ArticleAdultmedicine.medical_specialtyCombination therapyHepatitis C virusAlpha interferonInterferon alpha-2Antiviral AgentsGastroenterology and Hepatology/HepatologyInternal medicineRibavirinInfectious Diseases/Viral InfectionsmedicineHumansRetrospective StudiesVirology/Antivirals including Modes of Action and ResistanceInfectious Diseases/Antimicrobials and Drug Resistancebusiness.industryRibavirinlcsh:RGenetic VariationInterferon-alphaMicrobiology/Medical MicrobiologyVirology/Mechanisms of Resistance and Susceptibility including Host Geneticsmedicine.diseasebiology.organism_classificationLogistic ModelschemistryImmunologylcsh:QbusinessPLoS ONE
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Identification of potential mycoherbicides using a metabarcoding approach

2018

National audience

[SDE] Environmental Sciencesmodes d’action[SDV]Life Sciences [q-bio]genetic diversityinteractions adventice-microorganismeslutte biologique[SDV] Life Sciences [q-bio]bioherbicidemicroorganism-weed interactionsdiversité génétiquemodes of action[SDE]Environmental Sciences[SDV.BV]Life Sciences [q-bio]/Vegetal Biology[SDV.BV] Life Sciences [q-bio]/Vegetal BiologybiocontrolComputingMilieux_MISCELLANEOUS
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The Role of Fc Receptors on the Effectiveness of Therapeutic Monoclonal Antibodies.

2021

Since the approval of the first monoclonal antibody (mAb) in 1986, a huge effort has been made to guarantee safety and efficacy of therapeutic mAbs. As of July 2021, 118 mAbs are approved for the European market for a broad range of clinical indications. In order to ensure clinical efficacy and safety aspects, (pre-)clinical experimental approaches evaluate the respective modes of action (MoA). In addition to antigen-specificity including binding affinity and -avidity, MoA comprise Fc-mediated effector functions such as antibody dependent cellular cytotoxicity (ADCC) and the closely related antibody dependent cellular phagocytosis (ADCP). For this reason, a variety of cell-based assays have…

modes of action (MoA)GlycosylationQH301-705.5medicine.drug_classCellReceptors FcReviewBiologyMonoclonal antibodyCatalysisInorganic Chemistrychemistry.chemical_compoundMonoklonaler Antikörper ; effector function ; antibody dependent cellular phagocytosis (ADCP) ; therapeutic monoclonal antibodies (mAbs) ; Fcγ receptor (FcγR) ; modes of action (MoA) ; antibody dependent cellular cytotoxicity (ADCC)medicineAnimalsHumansAvidityClinical efficacyBiology (General)Physical and Theoretical ChemistryReceptorQD1-999Molecular BiologySpectroscopyAntibody-dependent cell-mediated cytotoxicityEffectortherapeutic monoclonal antibodies (mAbs)Organic ChemistryAntibody-Dependent Cell CytotoxicityAntibodies Monoclonalantibody dependent cellular phagocytosis (ADCP)General MedicineFcγ receptor (FcγR)Computer Science ApplicationsImmunoglobulin Fc Fragmentsantibody dependent cellular cytotoxicity (ADCC)Chemistrymedicine.anatomical_structurechemistryImmunologyImmunotherapyeffector functionInternational journal of molecular sciences
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